Then we did observe the “”omental ball”" of 18 × 13 × 6 cm in size, which was suspended by a pedicle twisted on its axis four times (Figure 3) and was easily resected. Our patient had an uneventful recovery and was discharged two days after. Histology Findings: omental pedicle of 18 × 13 × 6 cm in size,

with lobed and cyanotic look. Evidence of hemorrhagic infarction and focal fat necrosis areas at the section. Microscopic: Omental tissue characterized by acute extensive hemorrhagic infarction and fat necrosis, with polymorph nuclear cells infiltration of vein vessels and focal necrosis areas. Figure 3 Example of POT. A normal MS-275 supplier appearing omentum was above the torsion point. You can see the vascular hanged

and the torsion point with the distal thickened Selleckchem JSH-23 and congested omentum. Discussion POT is a rare pathological selleck compound condition which presents with generic symptoms, and may mimic a variety of acute abdominal conditions such as cholecystitis, acute diverticulitis, appendicitis [6] and Meckel diverticulum [7]. The pathogenesis of POT with infarction has not been established, however, some anatomical malformations and anomalies are recognized as predisposing factors to OT: presence in the great omentum of tongue-like projections and bifid and accessory omentum, anomalous vascular blood supply, other vascular anomalies that modify the weight of the omentum, vascular kinking, irregular omental pad, mostly in obese patients [8, 9]. SOT

is more common than POT GNA12 and is associated with pre-existing abdominal pathologies, including cysts, tumours, foci of intra abdominal inflammations [10] and surgical wounds or scarring and hernial sacs [11]. Most cases of SOT occur in patients with inguinal hernia as reported by Morris et al. [2]. Mentioned in literature as precipitant factors are trauma of the abdominal wall, coughing, effect of lifting, bicycle racing, hard labour, ingestion of heavy meals, hyperperistalsys, violent purgation or the taxis of an hernia, causes of passive displacement of the omentum [12]. The OT determines the omentum twists around a pivotal point, usually in a clockwise direction. Engorgement of the tortuous veins that are more easily compressed may compromise venous return, and the distal omentum becomes congested and oedematous. Recovery may follow or the process may go on [2]. Resultant hemorrhagic extravasations create a characteristic serosanguineous fluid inside the great omentum and in the peritoneal cavity. As the torsion progresses, arterial occlusion leads to acute hemorrhagic infarction and eventual necrosis of the omentum occur [6].